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Protective Effects of Glucagon Like Peptide-1 on HIT-T15 beta Cell Apoptosis via ER Stress Induced by 2-deoxy-D-glucose. |
Ju Young Kim, Seong Kyu Lee, Haing Woon Baik, Ki Ho Lee, Hyun Jin Kim, Kang Seo Park, Byung Joon Kim |
Diabetes Metab J. 2008;32(6):477-487. Published online December 1, 2008 DOI: https://doi.org/10.4093/kdj.2008.32.6.477 |
Protective Effects of Glucagon Like Peptide-1 on HIT-T15 β Cell Apoptosis via ER Stress Induced by 2-deoxy-D-glucose The effects of glucagon-like peptide-1 on the beta cell α-lipoic acid inhibits high glucose-induced apoptosis in HIT-T15 cells Emulsified isoflurane alleviates rat islet beta cell RIN-m5F apoptosis induced by glucose via inhibiting endoplasmic reticulum stress Exendin-4, a glucagon-like peptide-1 receptor agonist, inhibits cell apoptosis induced by lipotoxicity in pancreatic β-cell line Artemisinin and dihydroartemisinin promote β-cell apoptosis induced by palmitate via enhancing ER stress Silk fibroin has a protective effect against high glucose induced apoptosis in HIT-T15 cells Effects of glucagon-like peptide-1 (GLP-1) on glucose homeostasis and food intake High glucose potentiates palmitate-induced NO-mediated cytotoxicity through generation of superoxide in clonal β-cell HIT-T15 Effect of the alpha-lipoic acid on apoptosis in HIT-T15 cells induced by high glucose |